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Symptomatology
of
theNeurological
DiseaseSection
1ConsciousnessConceptConsciousness
is
awarenessof
the
body
toself-state
andthe
environment
.Quantitative
changes
ofconsciousness
mean
reducedalertness
:SomnolenceSoporComasomnolenceIn
the
early
stageIn
the
state
of
continuedsleepingCan
beawakedTalk
correctly
and
can
completethe
examination
after
awakingFall
in
sleep
after
stoppingstimulussoporIn
the
state
of
continuedsleepingCan
be
awaked
only
by
severepain
and
heavy
soundTalk
simply
and
uncompletelyafter
awaked,few
spontaneousspeechFall
indeep-sleep
after
stoppingstimuluscomaIs
the
mostseriousunconsciousnessComplete
loss
of
consciousnessNo
reaction
to
all
kinds
ofstrong
stimuliNo
active
movement
and
can’topen
his
eyes
spontaneouslyClassification
of
comaSuperficial
comaMiddlecomaDeepcomaSuperficial
coma1.
loss
of
consciousness
(noresponse
and
awaked)2.
reactive
to
stimulus
(pressingorbit)3.
spontaneous
action4.
have
corneal
reflex,
pupilreaction,
tendon
reflex5.
no
change
in
lifesigns(BP,P,R,T)Middle
coma
:betweensuperficial
coma
and
deep
comaDeepcoma1.
loss
of
consciousness
(noresponse
and
awaked)2.
no
reactive
to
stimulus(pressing
orbit)3.
completely
loss
of
spontaneousaction4.
loss
of
reflex,
pupil
reaction,tendon
reflex5.
having
change
in
life
signsLocked-in
syndrome
is
arareneurological
disordercharacterized
by
completeparalysis
of
voluntary
musclesinall
parts
of
the
body
(except
forthose
that
controleyemovement).The
lesion
site
is
inthe
base
of
the
pons.
It
mayresult
from
traumatic
braininjury,
vascular
diseases,demyelinating
diseases,
ormedication
overdose
.Individuals
withlocked-in
syndrome
areconscious
and
have
cognitive
function,
butare
unable
to
speak
or
move.
The
disorderleaves
the
patient
completely
mute
andparalyzed.
Communication
may
be
possiblewith
blinking
eye
movements
.Section
2Aphasia
,
ApraxiaandAgnosiaBroca
aphasia
:
is
also
called
motiveaphasia
or
expressive
aphasia.Thelesion
is
in
Broca
area.It
ischaracterized
by
oral
expresssingdysfunction.The
ability
ofrepeat,named,read
and
write
hasbeen
injuried
on
different
degrees.Wernicke
aphasia
:
is
also
calledauditory
aphasia
or
sensoryaphasia.The
lesion
is
in
Wernickearea.It
is
characterized
by
severecomprehension
dysfunction.
Theability
of
repeat,named,read
and
writehas
been
injuried
on
different
degrees.Com竭pl桐e雪tea垂起ph臭asi量aIsa独l知so性箭cal已l落ed私mi化x杰eda昼庭ph期asi和a.惹It晓i典st号雨he聋m旷os
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art饥i惩cul染ar母f习i叛nge发r,傻d突is些ti县n蕉gui气sh愈t莫he弦l伍ef发ts闭i边de头橡and散r划i锣ght瘦s都i捎de,劈燕李so吨met淋i筹mes窗r炼e茂adSec床ti刚on树3此Se疫izu碎r基e阴&S舱yn季co速p请eThe炒d器if达fe关r泄ent盯ia骂l乡丰po腿in藏to萄f扮山sei退zu果r舟ea编nd湖作sy丽nc虾ope便Clinical
featuresseizuresyncopeomenNo
or
short
(several
s’)long)relationship
to
positionnoUsually
when
standLasting
timeDay
and
night
,more
in
sleepMore
in
daySkincolorGreen
purple
or
normalpaleLimb
convulsionmorerarelyConfusion
after
attackoftenNone
or
rarelyNeurological
signhavenoCardiac
dysfunctionnooftenEEG
abnormaloftenrarelySec营ti畅on笨5殊Nys竿ta超gm触us知
Cl莫ass沾if窝i似edi治n但to捧:追ju土mpi与ng衔牌nys描t裁ag否mus昼clo掠ck来-l颗i港ken告y桃st投a闯gm乳usnys券t驾agm同u际sCl躲ass耗if册i姨edi计n会to兰:设ho泄ri袍z摩ont匙al吸刃nys只t绕ag鞭mus谷ver径ti痒ca研l挡nys乔t典ag紫mus送rot便at多en遵y冒st璃a接gm脑usSec捏ti症on量6阔Par盲a蓬ly巡寿si款sDif艘fe番r怒ent踢ia筋ld午i家a胀gn尸osi雕sfeaturesSpastic
paralysisFlaccid
paralysisdistributionwide,
the
wholelimbLocal(muscle
group))Muscle
tensionincreasedreflexTendon
reflex
increased,superficial
reflex
disappearordecreasedTendon
feflex
decreaseddisappearsuperficial
reflex
disappearPathologicalreflex(+)(-)Muscle
atrophynohaveMusclefasciculationnohaveSkinnutritiondysfunctionnoOften
haveEMGvelocity
isNerve
conduction
velocity
is
abnormalMuscle
biopsyNerve
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