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General
PrinciplesoftheDiagnosisAndManagement
ofFRACTURETheFirstAffiliatedHospital,ChongqingUniversityofMedicalSciencesDepartmentofOrthopedicsHuangweiEpidemiology
theUnitedStates
Traumacausesmorethan140,000deathsperyearleadingcauseofdeathforthoseaged1-34yearsmorethancoronaryarterydisease,cancer,andstrokecombinedbeforeage65yearsIn2000,morethan50millionAmericansunderwentmedicaltreatmentforaninjury.Theestimatedlifetimecostoftheseinjuriesisbelievedtobe$406billion.DEFINITIONofFRACTUREThecontinuityandcompletenessof
bonehavebroken
Afractureisacompleteorincompletebreakinthecontinuityofabone
TerminologyAnatomy:Thefractureisdescribedwithrelationtothebonesinvolvedandthelocationwithinthebone(diaphysis,metaphysis,physis,epiphysis).Articularsurfaceinvolvement:Doesthefracturehaveintra-articularinvolvement?Isthereintra-articulardisplacementorgapping?Displacement:Isthedistalfracturefragmentdisplacedcomparedwiththeproximalfragment?Towhatdegreeorpercentageisthefracturedisplaced?Angulation:Theangulardeformityisdefinedindegreesintermsofthedistalfragmentinrelationtotheproximalfragmentorwithrespecttotheproximalapexofthedistalfragment.Rotation:Rotationaldeformityisdescribedbothclinicallyandradiographically.Shortening:Hasthefracturecausedshorteningoftheinvolvedbone?Towhatextenthasshorteningoccurred?TerminologyFragmentation:TheMullerAO(ArbeitsgemeinschaftfürOsteosynthesefragen[AssociationforOsteosynthesis])ComprehensiveClassificationofFracturesprovidesastandardizeddescriptionoffracturepatterns,makingcommunicationregardingsuchinjuriesmorepreciseandunderstandable.Amultifragmentaryfractureisonethathasseveralbreaksinthebone,creatingmorethan2fragments.Wedgefracturesareeitherspiral(lowenergy)orbending(highenergy)andallowtheproximalanddistalfracturefragmentstocontacteachother.Thecomplexmultifragmentaryfractureisasegmentalfractureoroneinwhichthereisnocontactbetweentheproximalanddistalfragmentswithouttheboneshortening.Simplefracturesarespiral,oblique,ortransverse.FactorsResponsibleforFractures
Directviolence
appliedtothebonealsodamagessurroundingsofttissueAtappingforceappliedtotheboneproducesanobliquefractureAcrushinginjuryresultsinafragmentedfractureoftheboneApenetratingdirectinjuryfromahigh-velocitygunshotblastdestroysboneandsofttissueFactorsResponsibleforFracturesIndirectviolence
appliedtotheboneproducessignificantlylessdamagetosoftandhardtissuesAnabductionorverticalormusclepullforcecausesacompressionoravulseorobliqueorspiralfractureFactorsResponsibleforFracturesManydiseasesofbonemaycausedestructionofboneormayweakentheboneproducesapathologicfractureinflammationtumorortumor-likediseasesosteogenesisimperfectametabolicdiseasesetalFactorsResponsibleforFracturesInrepeatedstresses,whichcausefatiguefracturesthesestressfracturearemostfrequentlyencounteredinboneofthelowerextremity:theneckofthesecondorthirdmetatarsalbonetheshaftofthetibiaorfibulartheneckofthefemurCLASSIFICATIONSFracturesmaybecausedby
ViolencedirectindirectInjuryfractureMusclepullAvulsionfractureFatigueStressfractureDiseasePathologicalfractureclassificationsFromWhetherHaveDirectorindirectCommunicationBetweentheFractureandWoundoftheSkinandMucosaFromWithinOpenFracture
FromWithoutClosedFractureDirectViolenceIndirectViolenceMusclepullAvulsionfractureOpenFractureOpenComminutedFracture车祸致左小腿开放性毁损OpenandComminutedfractureclassificationsFromStability
Stable
TransversefractureGreenstickfractureImpactedfractureEpiphysealinjuryTransversefractureofthelefthumerusImpactedfractureoftheneckoftherightfemur
FromStability
Unstable
SpiralfractureObliquefracture
Slopeofmorethan200o0ComminutedfractureAvulsionclassificationsThecomminutedfractureoftherighthumerusThelongspiralfractureoftheleftfemurdistalGreenstickTransverseAngulationObliqueSpiralDoubleComminutedImpactedCrushAvulsionInvolvingajointCompressionFracture-DislocationEpiphysealinjuryclassificationsPatternofDisplacementAngulationRotationLateralSideOverlaporShorteningImpactingMechanismofthedisplacement
ofthefragmentsDirectionoftheviolenceEffectsofgravityMusclepullImproperFirstAid肌肉拉力造成的骨折移位CLINICAL&RADIOLOGICAL
FEATURESOFTHEFRACTURESystemicManifestationsPrimaryShock(NeurogenicShock)SecondaryShock----Haemorrhagia(-ge)Notice:excludetocerebralinjuryandrespiratoryembarrassmentCLINICAL&RADIOLOGICAL
FEATURESOFTHEFRACTURESystemicManifestationsLowFeverorSlightFevertemperature380C
traumareactionhaematoma
absorptionWBC(whitebloodcell)ESR(erythrocytesedimentationrate)CLINICAL&RADIOLOGICAL
FEATURESLocalManifestationTraumaticInflammation:PainLocalSwellingLossorImpairmentofFunctionEcchymosisLocalizedbonetenderness
SpecificSigns
ThefollowingfeaturesarepathognomonicoffractureDeformityAbnormalMobilityCrepitusorGratingCLINICAL&RADIOLOGICAL
FEATURESAbsenceofdysfunctiondoesnotprecludefracturethisisparticularlytrueof:greenstickfacturesimpactedfracturesfatiguefracturesCLINICAL&RADIOLOGICAL
FEATURESRadiologicalExaminationFractureyesorno?Patternoffracture?Anatomicpartofthefracture?SelectingofTreatmentMethods!COMPLICATIONS
OFFRACTUREEarlyPeriodShockFatembolismInjurytoImportantOrganandTissue(nervousandvascular)Osteo-fascialCompartmentSyndrome
骨筋膜室综合征
Osteofascial
CompartmentSyndrome早期并发症晚期PainPainlessPallorParesthesiaParalysisPulselessness早期PainDull-redPassivemotionpainPulsenessOsteofascial
CompartmentSyndrome肱骨髁上骨折致肱动脉损伤、骨筋膜室综合征COMPLICATIONSOFFRACTURE
EarlyPeriodInfection:tetanus;gasgangrene;osteomylitisThromboembolism:deepvenousthrombosisandpulmonaryembolismAcuterespiratorydistresssyndromeMultiple-organdysfunctionsyndromeCOMPLICATIONOFFRACTURELaterPeriodSystemicComplicationPressureSoresPneumoniaInfectionoftheBladderCOMPLICATIONSOFFRACTURELocalComplications
MyositisOssificansPost-traumaticOsteoarthritisJointStiffnessReflexSympatheticDystrophySudeckatrophyAvascularNecrosisofTheBoneIschemicContracture(Volkmann’s)COMPLICATIONSOFFRACTURE
LaterPeriodDelayedunionNonunionMalunionorangulardeformitiesShorteningGrowtharrestGrowthstimulationFRACTUREHEALING
OrganandTissue
Scar
Repair
Fracture
RegenerationNewBone
Stagesofbonehealing1.Forthefirst2weeks,bonehealingfollowsthesamepattenasthehealingofskinoranyortherwound.Thesiteofthewoundisfilledwithbloodandthebrokenendsofbonebecomenecrotic.2.Thebloodclotisinvadedbymacrophagesandosteoclastswhichremovedeadboneandosteoblastswhichproducebone,insteadofthefibroblastswhichformfibroustissueinsofttissueinjuries.3.Beteen2to6weeksafterinjuryosteoidtissuedevelopsandformsafirmmass,orcallus,aroundthefractureandossificationoftheosteoidbegins.Callusformsbothoutsidetheboneassubperiostealcallus,andinsideasendostealcallus.ThepHofthetissuesinreasesatthisstageandcalciumisdeposited.4.Between6and12weeks,ossificationoccurs,asolidbonybridgecrossesthegapandtheboneregainssomemechanicalstrength.5.Between12and26weeks,thecallusmatures.6.Between6and12months,thegapsbetweenthecorticalendsarebridged.7.Between1and2years,remodellingoccurs,bonyprominencesbecomesmoothandnormalbonearchitectureisrestored.Thetimingisveryvariableandismuchfasterrinchildren,inwhomcalluscanbeseenat2weeks.FRACTUREHEALING
StagesofFractureHealing1.Haematoma→→Fibrosis(2w)↓Fibrinogen+ReticularFibril→BloodClots↓NewVessels+MesenchymalCellInflammatoryCell→GranulationTissue
FRACTUREHEALING2.PrimaryCallusFormation(6-8w)OriginoftheCallusCellsDOPC(DeterminedosteogenicPrecursorCells)IOPC(InducibleOsteogenicPrecursorCells)FRACTUREHEALINGOriginoftheCallusMineralDeadBoneOsteoblasts,Chondroblasts↓↓CO2↑Phosphorylase↑↓↓pH↓HydrolyzetoPhosphate(intheHematoma)(inthePlasma)FRACTUREHEALING↓↓CalciumofFragmentPhosphateFreeDissolved↓Heamatoma→CalciumPhosphate→DeposittoBoneMatrix→
Callus(WovenBone)
FRACTUREHEALINGEndo-chondralOssificationIntra-membranousOssificationNewboneformseitherthroughappositionalossificationwithoutinitialcartilageformationorbyenchondralossificationofapreminaryfibrocartilagecallus.Inbothinstances,theossificationprocessisintimatelyrelatedtorevascularization,andtheactiveosteoblastsappeartobederivedfromeitherthewallsofthesmallvesselsorfromcirculatingbloodcells.Bonerepairtakesplaceintheperiosteal,cortical,andmedullaryregions,butmostoftherevasculaizationofafractureisfrommedullarycirculation.Whichoftheseareaspredominateintherepairprocessdependsonthenatureofthebone,thedegreeofinitialinjury,andtheamountoffractureimmobilizationduringhealing.Themostrapidofalltheprocessesofhealingistheexternalorperiostealcallus,whichpredominatesinfracturetreatednonoperativelyandwithearlymusclefunction.Itdependsprimarilyonsurroundingsofttissuebloodsupply.Thiscallusisquitetolerantofcontrolledfracturemotion;infactitismostinevidencewhenfracturemotionoccurs,e.g.,inribfractures.Asecondprocessislatemedullarycallus,whichpredominateswhentheexternalcallushasfailed.Itisassistedbyrigidimmobolizationandisthepredominanthealingprocesswithcompressionplatefixation.Intramedullarycallus,asMcKibbonhaspointedout,isnotanevanescentburstofactivitybutaprocessthatseemstopursueitsgoaloffracturebridgingrelentlessly.Thethirdprocessisthatofprimaryboneunionthatexplainstherarephenomenonofhealingwithoutexternalcallus.Itdependsonthemechanismofboneturnover,whichisoccurringatalltimesandwhichcanrespondtobridgefracturedbonecortices,providedthattheyarerigidlyimmobilized.Bythisprocessthedeadcorticalboneimmediatelyadjacenttothefractureisinvadedbynew,longitudinallyorientedosteonesfromtheneighboringlivebone.Themajordisadvantageofprimaryboneunionisitsgreatslownessanditsdependenceonrigidimmobilization.Inmostinstanceswithcompressionplating,thefracturegapissufficientlylargethatitfillsbyboneformedthroughappositionalgrowthinwardfromtheexternalperiostealsource.Thisprimaryboneformsrapidlyinthe4-6weeksbuthasrelativelypoorattachmenttotheavascularboneends.Thisprimarycallusmustberemodeledandreplacedat6-8weeksbysecondaryosteonesbridgingthefracturegaplongitudinally.Theentireprocessofhealinginboneaswellasinsofttissueisdependentontheprocessofrevascularization.FRACTUREHEALING3.Remodelling(8w---Someyears)ModellingWovenBoneLamellarBone(8---12wClinicalHealing)Remodelling(12w---SomeYears)
Wolff’sLawStressandPiezoelectricityFRACTUREHEALINGFRACTUREHEALINGFRACTUREHEALING
CriterionofFractureHealing
1.AbsenceTendernessandAxial
PercussionPain2.AbsenceAbnormalMobility3.VisibleContinuedCallushasBridgedFragmentsandFractureLineIndistinctFRACTUREHEALING4.AftertheSplintshasbeenRemoved,A.TheArmisParalleltoGround,andHold1kgPersistin3min.B.TheLegsisWalk30Paceon3min.C.AfterObserve2wContinually,GoodResulthasShowedontheFractureSiteFRACTUREHEALING
CriterionofFractureHealingINFLUENCEDFACTORS
ofFRACTUREHEALINGSystemicFactors1.Age2.SystemicCondition
LocalFactors1.PatternofFracture
INFLUENCEDFACTOR
ofFRACTUREHEALING2.
BloodSupply(Fragment)A.AllgoodBothFragmentsB.OnegoodandOtherBadC.AllBadBothFragmentsD.AbsenceBloodSupplyontheFragmentINFLUENCEDFACTORS
ofFRACTUREHEALING3.Infection4.InjuriesofSoftTissue5.InterpositionofSofttissueintoFractureGap
6.BasicDisease(local)INFLUENCEDFACTORS
ofFRACTUREHEALING
MethodofTreatment?!1.RepeatedReductionbyManipulation2.OverTractionofFragments3.NoCorrectFixation4.NoCorrectDebridement5.InfluenceofOpenReduction6.NoCorrectFunctionalExercisesINFLUENCEDFACTORS
ofFRACTUREHEALING
BiologicalFactors20%MedicinalFactors80%!!
FIRSTAIDofFRACTURE
PurposeofFirstAid1.SavetheLive2.PreserveLimbs3.SafetyQuicklyTransport
SystemicConditionShockComaRespiratoryEmbarrassmentFIRSTAIDofFRACTURETemporaryCareofFracture
OpenClosed
CorrectTransportPRINCIPLESofFRACTURE
TREATMENTReduction
ImmobilizationRehabilitationMedicationREDUCTION
CriterionofReduction1.AnatomicReduction2.FunctionalReduction
(1).NoObserveGapandRotation(2).Shortening<1~2cmintheLeg(3).Angulation<15~10(4).AppositionoftheFragments>
1/3(shaft),or3/4(epiphyseal)REDUCTION
MethodsofReduction1.ByClosedManipulation2.ByMechanicalTractionwithorwithoutManipulation3.ByOpenReductionIMMOBILIZATION
ReasonsofImmobilization1.PreventionofDisplacementorAngulation2.PreventionofMovement3.ReliefofPainIMMOBILIZATIONMethodsofImmobilization1.ByWoodenSplintorPlasterorSplint(other)2.ByContinuousTraction3.ByExternalFixer4.ByInternalFixationREHABILITATION1.ActiveMuscularContraction(AfterInjuryWithin2Weeks)2.ActiveExercisesJointsofAdjacentFracture(AfterInjury2~8w)3.ActiveResistanceExercises(After8w)4.PhysiotherapyMedication
TraditionalMedication
ForbidUsingNonsteroidAnti-InflammatoryDrugs(NSAIDs)TREATMENTofOPEN
FRACTURE
PreventingInfection!!
TREATMENTofOPEN
FRACTURE1.TypeofOpenFractureFromWithinFromWithout
Potential
TREATMENTofOPEN
FRACTURE
2.ConditionsoftheWound
ContaminativeExtentDegreeofSoftTissueInjuryTREATMENTofOPEN
FRACTURE3.DebridementSamePrinciplesofSurgicalDebridementGoldenTime:6~8h(afterinjury)LargerBonyPieceShouldbePreservedSelectingMethodsofFixationRepairofNerve、Tendon、VesselSkinClosureFEATUREofOPENI
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